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Iron accumulation, a hallmark of ferroptosis, can be modulated by NRF2-regulated genes involved in iron storage and transport [40]

This antibody specifically recognizes the mono-methylation of R169 on both exogenous and endogenous UBE2m, which was elevated in COM-treated HK-2 cells compared to that in the NC group and responded to PRMT1 manipulation (Fig

FIGURE 2 3.5 Expression levels of pro-inflammatory genes in the kidney tissue As depicted in Figure 3, AFT could trigger renal inflammation indicated by considerable upregulation of mRNA expression levels of the pro-inflammatory cytokines (TNF-, IL-1, and IL-6) compared with controls